Quantifying T lymphocyte turnover
From MaRDI portal
Publication:745045
DOI10.1016/J.JTBI.2012.12.025zbMATH Open1322.92018OpenAlexW1971931476WikidataQ36806548 ScholiaQ36806548MaRDI QIDQ745045FDOQ745045
Authors: Alan S. Perelson, Rob J. De Boer
Publication date: 13 October 2015
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: http://europepmc.org/articles/pmc3640348
Recommendations
- Parameter identification for model of T cell proliferation in lymphopenia conditions
- Modeling the lifespan of human \(T\) lymphocyte subsets
- Parameters identification for a model of T cell homeostasis
- Dynamics of T cell activation threshold tuning
- Modeling of T cell population development and estimation of resource allocation effectiveness
- scientific article; zbMATH DE number 596334
- Estimating lymphocyte division and death rates from CFSE data
- An age-structured model for T cell homeostasis in vivo
- A phenomenological approach to the dynamics of activation and clonal expansion of \(T\) cells
Cites Work
- On the formulation and analysis of general deterministic structured population models. II. Nonlinear theory
- An Algorithm for Least-Squares Estimation of Nonlinear Parameters
- Title not available (Why is that?)
- Decline in excision circles requires homeostatic renewal or homeostatic death of naive T cells
- Modeling T cell proliferation and death in vitro based on labeling data: generalizations of the Smith-Martin cell cycle model
- Modeling deuterated glucose labeling of T-lymphocytes
- Solving Ordinary Differential Equations I
- Estimating division and death rates from CFSE data
- Opportunistic infection as a cause of transient viremia in chronically infected HIV patients under treatment with HAART
- Estimating lymphocyte division and death rates from CFSE data
- Title not available (Why is that?)
- Analysis and simulation of division- and label-structured population models. A new tool to analyze proliferation assays.
- Estimation of cell proliferation dynamics using CFSE data
- Scaling aspects of lymphocyte trafficking
- Determining the expected variability of immune responses using the cyton model
- A Stochastic Model to Analyze Clonal Data on Multi‐Type Cell Populations
- Models of CD8+ responses: 1. what is the antigen-independent proliferation program
- A general mathematical framework to model generation structure in a population of asynchronously dividing cells
- Evaluation of multitype mathematical models for CFSE-labeling experiment data
- Computational analysis of CFSE proliferation assay
- Interpreting CFSE obtained division histories of B cells in vitro with Smith-Martin and cyton type models
- A Mixture Model With Dependent Observations for the Analysis of CSFE–Labeling Experiments
- A division-dependent compartmental model for computing cell numbers in CFSE-based lymphocyte proliferation assays
- The rescaling method for quantifying the turnover of cell populations
- Mathematically modeling dynamics of T cell responses: predictions concerning the generation of memory cells
- Saddlepoint approximations to the moments of multitype age-dependent branching processes, with applications
- The effect of correlations on the population dynamics of lymphocytes
- A division-dependent compartmental model with cyton and intracellular label dynamics
- A composite likelihood approach to the analysis of longitudinal clonal data on multitype cellular systems under an age-dependent branching process
Cited In (26)
- Dynamics of HIV infection in lymphoid tissue network
- Germinal center dynamics during acute and chronic infection
- A mathematical model for a T cell fate decision algorithm during immune response
- Caspase-1-mediated pyroptosis of the predominance for driving \(\mathrm{CD}4^+\) T cells death: a nonlocal spatial mathematical model
- A mathematical model of the effects of aging on naive T cell populations and diversity
- Optimal control of an HIV model with a trilinear antibody growth function
- A fit of CD4\(^+\) T cell immune response to an infection by lymphocytic choriomeningitis virus
- Pattern formation in a nonlocal mathematical model for the multiple roles of the TGF-\(\beta\) pathway in tumour dynamics
- A minimal model of T cell avidity may identify subtherapeutic vaccine schedules
- Modelling and investigation of the \(\mathrm{CD}4^+\) T cells -- macrophages paradox in melanoma immunotherapies
- Insights into the mechanisms of thymus involution and regeneration by modeling the glucocorticoid-induced perturbation of thymocyte populations dynamics
- Title not available (Why is that?)
- Inferring average generation via division-linked labeling
- How many TCR clonotypes does a body maintain?
- Predicting pathogen-specific CD8 T cell immune responses from a modeling approach
- Dynamics of an HIV infection model with virus diffusion and latently infected cell activation
- The SEIR model with pulse and diffusion of virus in the environment
- Mathematical models for CFSE labelled lymphocyte dynamics: asymmetry and time-lag in division
- Adaptive protocols based on predictions from a mechanistic model of the effect of IL7 on CD4 counts
- Dynamics of T cell receptor distributions following acute thymic atrophy and resumption
- Predicting and explaining with models: a few remarks on mathematical immunology
- Mathematical investigation of innate immune responses to lung cancer: the role of macrophages with mixed phenotypes
- Quantitative immunology for physicists
- Mathematical models for immunology: current state of the art and future research directions
- Sample path properties of the average generation of a Bellman-Harris process
- Dynamics of a PDE viral infection model incorporating cell-to-cell transmission
This page was built for publication: Quantifying T lymphocyte turnover
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q745045)